You're wrong my friend. There is everything wrong with fructose; as I have pointed out previously, fructose is metabolised completely differently to other sugars, in essence it is sent straight to the liver and converted to fat and is chiefly responsible for causing insulin resistance in the liver.
Oh yeah. So where did you get that from?
High sugar intake inhibits the conversion of fat into useable fuel carbohydrates. Little or none of it is converted into fat. Get your facts right before you accuse someone else of being wrong.
Most of the fat in our bodies comes from fat content in diet. The liver produces fatty substances such as chloresterol and there is a purpose behind that. But it doesn't convert fructose into fat. Another thing is that insulin resistance causes raised blood sugar levels, it's not necessarily a consequence of it.
High sugar intake inhibits the conversion of fat... No shit Sherlock. Of course it does, I didn't say it didn't but you are being less specific on the type of sugar because as I said! fructose is metabolised in the liver and does not affect blood sugar levels in the same way as say Glucose does. It stands to reason that it you are consuming sugar then the body had little need to convert stored fat to glucose. Insulin resistance doesn't cause raised blood sugar levels, it simply means that the body is less able to deal with high blood sugar levels that occur as a result of dietary intake.
Most of our body fat comes from eating fat in our diet does it? So where did you get that from, a men's health magazine? You are no doubt one of the eat less fat brigade, when fat in our diet is not the problem.
Here are two direct references from an academic paper posted on the American society for clinical nutrition website... But note in particular the last comment that hepatic metabolism of fructose favours lipogenesis.
Fructose is an intermediary in the metabolism of glucose, but there is no biological need for dietary fructose. When ingested by itself, fructose is poorly absorbed from the gastrointestinal tract, and it is almost entirely cleared by the liver—the circulating concentration is ≈0.01 mmol/L in peripheral blood, compared with 5.5 mmol/L for glucose.
Fructose is metabolized, primarily in the liver, by phosphorylation on the 1-position, a process that bypasses the rate-limiting phosphofructokinase step (4). Hepatic metabolism of fructose thus favors lipogenesis, and it is not surprising that several studies have found changes in circulating lipids when subjects eat high-fructose diets
Perhaps also watch one of Dr Robert Lustigs Youtube lectures on why fructose is bad.
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